 |
| Author | Post |
|---|
Dr Trevor Marshall
Research Team
|
Posted: Wed Apr 18th, 2007 00:04 |
|
John,
I am glad Tickbite is helping guide you through the maze, as I am really busy right now. But I need to suggest that you get a copy of the DVD containing the presentation I gave to the University of Melbourne last November. Copies are available from the Australian Autoimmunity Foundation. At the moment you are only scratching the surface of what you need to know in order to understand the disease processes leading to ASD, and after you listen carefully to the DVD you should have a better overview. Tickbite can then guide you from that point.
|
Meg Mangin R.N.
Research Team
|
Posted: Wed Apr 18th, 2007 09:54 |
|
There is a lot of information in CELL WALL DEFICIENT BACTERIA AND THE MARSHALL PROTOCOL
You may be interested to read Why has there been a sharp rise in the incidence of Th1 inflammatory diseases?
|
tickbite
Member
|
Posted: Wed Apr 18th, 2007 13:44 |
|
I don't want you to not buy the DVD because it's really awesome, however
Nov. 12-21, 2006 Australian lecture tour to physicians at:
- John Curtin School of Medical Research, ANU
- Bio21 Institute Seminar (see below for video)
- Royal Perth Hospital
- Murdoch University
A pdf of Dr. Marshall's slides is available at http://www.autoimmunityresearch.org/tm_aust_2006.pdf
=======================================
The video of Dr. Marshall's presentation at Bio21 is now online at URLs:
http://autoimmunityresearch.org/bio21.ram
and
http://autoimmunityresearch.org/bio21b.ram
and
http://autoimmunityresearch.org/bio21a.ram
American Academy of Environmental Medicine conference on "The Brain and the Environment"; Oct. 26-29, 2006 in Hilton Head, SC- Plenary Sessions Syllabus, 41st Annual Meeting A New Approach to Treating Intraphagocytic CWD Bacterial Pathogens in Sarcoidosis, CFS, Lyme and other Inflammatory Diseases. (Certified for physician CMEs)
The excellent presentation is available online in RealVideo 9 format....you can view it from URL
http://autoimmunityresearch.org/aaem_2006_mirror1.ram
or from URL
http://autoimmunityresearch.org/aaem_2006_mirror2.ram
or from URL
http://autoimmunityresearch.org/aaem_2006.ram
A DVD of the AAEM presentation is also available at
http://autoimmunityresearch.org/
A transcript of the AAEM presentation is available at
http://www.carouselcharts.com/TranscriptionAAEM.pdf
And there's a couple more, but these are very good. I copied and pasted from here;
http://www.marshallprotocol.com/forum2/2274.html
"papers and presentations..."
Also, there is much question that Dr. M raises about CWD in the vaccinations themselves.....Hope you like Dr. M's presentations!
____________________
"Lyme","CFS", Meningitis
Phase3 8-2-07, MP on hold 11/2007
|
healingjason
Member in Phase 3
|
Posted: Mon Apr 23rd, 2007 23:05 |
|
Meg Mangin R.N. wrote: There is a lot of information in CELL WALL DEFICIENT BACTERIA AND THE MARSHALL PROTOCOL
You may be interested to read Why has there been a sharp rise in the incidence of Th1 inflammatory diseases?
Meg
On your second citing/link, I note my boy was exposed to at least 3 of the risk factors, namely:
1.too many beta-lacatam abxs;
2.vaccinations, including Hep b shots; and
3. infant formula (Jason was bottle fed).
being an infant, he would not have had excessive exposure to the sun.
This analysis suggests more autism in Western countries than in less developed countries. To my knowledge, there is no data to test this.
However, I note autism first emerged in the wealthy Western classes in the 1930s and it was believed to be a product of 'refrigerator' mothers (nannies did the child rearing) until debunked in the 1960s (by a Dad of an autistic child, Bernard Rimland). To explain the greater presence of autism in wealthy families, it has since been suggested that vaccinations were most taken up in this population segment and it has been the Hg in the vaccines that has caused the autism. The idea that wealthy families would also have been more likely to have used abxs and adopted formula feeding has not been considered. This may be a better explanation of the rise in autism numbers in the West than has hitherto been posited.
John
____________________
Father of Jason, 11 year old autistic boy. Chronic infection evident from urinary amino acids (see bioscreenmedical.com). Benicar 40 mg QW8H from 7 Apr 07. Phase 3 from 28 June 08. Covered up, sunscreen.
|
tickbite
Member
|
Posted: Tue Apr 24th, 2007 11:36 |
|
Will heavy metals, toxins or mold hinder my recovery on the MP?
"red herring" indeed.....
____________________
"Lyme","CFS", Meningitis
Phase3 8-2-07, MP on hold 11/2007
|
healingjason
Member in Phase 3
|
Posted: Wed Apr 25th, 2007 20:42 |
|
Tickbite
From your link Will heavy metals, toxins or mold hinder my recovery on the MP?, I have snipped this quote from Trevor below:
"Inflammation increases copper and decreases zinc. This is because the inflammatory process results in an accumulation of zinc-containing proteins in the liver and increased liver synthesis of ceruloplasmin, which is the copper-binding protein."
A big deal for the Pfeiffer Institute which has promoted detoxification treatments for autism is their fairly ubiquitous finding among autistics of low blood zinc and high blood copper. However, I believe they attribute this to a heavy metal detoxification weakness, specifically, a deficiency of metallothionene (MT) which somehow elevates copper and reduces zinc. Their supplementation therapy aimed at improving MT levels and the copper/zinc balance has gained a lot of traction in the autism community and seen many dollars spent on their supplements.
Another red herring?
BTW, Dr Bill Woods of the Pfeiffer Institute claims to have shown that Beethoven's various ailments were caused by lead poisoning from an examination of a lock of Beethoven's' hair which was kept for many years and finally examined some few years ago. 'Twas a good documentary shown in Australia recently.
While I have yet to read up on the MP material on this, would I be being presumptious to suggest the high copper/low zinc finding is another indicator of chronic Th1 infection driving autism.
John
____________________
Father of Jason, 11 year old autistic boy. Chronic infection evident from urinary amino acids (see bioscreenmedical.com). Benicar 40 mg QW8H from 7 Apr 07. Phase 3 from 28 June 08. Covered up, sunscreen.
|
tickbite
Member
|
Posted: Thu Apr 26th, 2007 12:15 |
|
In my limited knowledge John, I know that MT is thought to only play a role in heavy metal detox and scavenging free radicals. However I also know that MT is induced by hormones and cytokines. MT also modulates cytokine response and for sure is part of many negative feedback mechanisms within the body. A multifunctional protein. I also know that it seems as though MT is directly induced by 1,25D. So there you have it, another indicator as you rightly put it. Deficieny, smyshunsee.........does sound more like a red-herring than a cause. If CWD forms can share genetic information (which they do) and parasitize our immune cells, then obviously they know how to take control of the cell and alter it's communication with the 'outside' world. As far as I can see it, bacterial colonies have an unlimited number of communication methods with which they use to induce genetic mutation within the cell causing it to wreak havoc. Bacteria are extremely diverse and maintain proliferative knowledge to a huge statistical degree.
As far as Beethoven.......could be lead poisoning! Most of the time though, it's never just one thing. I love Beethoven.
~Greg
____________________
"Lyme","CFS", Meningitis
Phase3 8-2-07, MP on hold 11/2007
|
wrotek
Member in Phase 3
| Joined: | Fri Dec 31st, 2004 |
| Location: | Wroclaw, Poland |
| Posts: | 1121 |
| Status: |
Online
|
|
Posted: Fri Apr 27th, 2007 14:18 |
|
http://archives.cnn.com/2000/HEALTH/children/07/18/autism.ap/
Look what i have read recently.
____________________
Lyme reflux chronic pain fatigue depression 125D36 Ph1Sep05 Ph2Oct06 Ph3Apr07 homebound in low lux NoIRs 25D<7 Oct06
|
healingjason
Member in Phase 3
|
Posted: Mon Apr 30th, 2007 20:35 |
|
Wrotek
I did a trial of vancomyicn over 4 weeks or so (some years back) for my boy to see if this would help. Unfortunately, there were no improvements.
There has been speculation that the children that were helped may have had Lyme and the vancomycin was effective against this.
John
____________________
Father of Jason, 11 year old autistic boy. Chronic infection evident from urinary amino acids (see bioscreenmedical.com). Benicar 40 mg QW8H from 7 Apr 07. Phase 3 from 28 June 08. Covered up, sunscreen.
|
healingjason
Member in Phase 3
|
Posted: Tue May 15th, 2007 00:01 |
|
All
This is new topic related to autism that folk may have views on.
This forum has discussed autism being a Th1 illness and explaining its rise - overuse of penicillin, vitamin D in formula milk, more contaminated vaccines etc.
One distinctive feature of autism is that 3 out of every 4 autistic people are male. I am not aware of any Th1 illness having a gender bias. Does any particular Th1 illness have a gender bias?
It is not obvious to me from what I have read on the MP that a Th1 illness would affect boys more than girls and render them autistic. My understanding is that a Th1 illness can strike anybody at any age and of any sex - CWD bacteria do not favour one type of host over another.
However, to posit that a Th1 pathology is the most likely cause of autism in children needs to explain why boys are more affected than girls. This is not discount that a Th1 illness can affect any specific child, including my son.
Mainstream medicine thinks autism is most likely a genetic condition as a greater percentage of siblings or twins have autism compared to unrelated children. However, this does not explain why boys are more affected.
The mercury people have an explanation for the gender bias. They assert that testosterone potentates the effect of mercury.
Can the MP theory explain why more boys suffer from autism than girls?
John
____________________
Father of Jason, 11 year old autistic boy. Chronic infection evident from urinary amino acids (see bioscreenmedical.com). Benicar 40 mg QW8H from 7 Apr 07. Phase 3 from 28 June 08. Covered up, sunscreen.
|
jcwat101
Research Professional
|
Posted: Tue May 15th, 2007 01:08 |
|
This is purely a conjecture and isn't really an "MP" explanation per se. But it may be that since autism affects the ability to relate socially, that the presence of estrogen is somewhat protective. It is my impression that women are, on average, more socially oriented and more in touch with their emotions (eg, statistics say women do actually talk, on average, a lot more than men).
There is a book out now on male-female differences, called "The Female Brain" and there may be something in it on the scientific basis for the differences that would relate to what I am saying.
So, my thought is that the bacteria may affect the boys and girls just as often, but the girls may develop other symptom complexes. This might include a slight "autistic tendency," but not enough to be diagnosed as such.
Joyce Waterhouse
Last edited on Tue May 15th, 2007 01:11 by jcwat101
____________________
20 yrs with CFS/FM/Lyme/IBS, food sensitivities; 1,25D/25D 8/04:64/11 1/05:22/6 9/05:1,25D=12 10/06:22/8, 4/07:25/<4 chewed Ben. 40mg q8h; Mod. P2: 2/23/05, P2: 4/06; P3: 1/1/07
|
tickbite
Member
|
Posted: Tue May 15th, 2007 13:18 |
|
healingjason wrote: Mainstream medicine thinks autism is most likely a genetic condition as a greater percentage of siblings or twins have autism compared to unrelated children. However, this does not explain why boys are more affected.
The mercury people have an explanation for the gender bias. They assert that testosterone potentates the effect of mercury.
Can the MP theory explain why more boys suffer from autism than girls?
Testosterone potentiating the effect of mercury sounds rather simple when you are looking at it from a genomic point of view. I don't pretend to know the answer to your question, but i'll make a few comments. We know that human cell genetic mutation occurs often from infectious pathology. What would happen if a stem cell were infected also? What would happen if male sperm were infected with CWD that transfers to an egg? In my idea of things at the moment, disease predisposition genetics of individuals are the way they are probably more so because of infectious pathology from parents than from random mutation. Now, i'm not discounting other mutation phenomena, just arguing a greater numbers game. As far as why boys are 3 out of 4..........it's up to you to figure out! My guess is those communities of DNA which begin austism have the genomic capability to manifest a specific signalling profile that may make males more susceptible due to innate biology. Each community of pathogens is never the same. Like the snow flake analogy. Slight differences in growth yield different characteristics upon the outcome. I think Trevor's ideas of neo-natal pathogens persisting in the brain are very important to the development of Th1 disease. It's very difficult to answer your question for me John, knowing that precise biological actions are complex, complex, complex. ~Grego
____________________
"Lyme","CFS", Meningitis
Phase3 8-2-07, MP on hold 11/2007
|
Claudia
Member in Phase 3
|
Posted: Wed May 16th, 2007 11:43 |
|
| Well, I've often seen autism described as a state of "extreme maleness".
____________________
MP Phase1 23Mar_06; Phase2 July 10_06; Phase3 Nov 4_06. Dx Thyroiditis (Thyroxine); arthritis; glaucoma; CFS (1988-92);Kidney & bladder probs. Feb06 1,25D=43.3; Aug07 1,25D=27.5; Feb06 25D=44; Aug07 25D=28; Nov07 25D=36; Mar08 25D=16.4
|
tibet
Member
| Joined: | Wed Mar 14th, 2007 |
| Location: | United Kingdom |
| Posts: | 71 |
| Status: |
Offline
|
|
Posted: Thu May 17th, 2007 12:22 |
|
hi
I work with autistic post 16's
Girls do get it too
stella
____________________
20 years CFS/ME Looking for helpful doctor in UK Have 3 grownup children with disease.
|
jcwat101
Research Professional
|
Posted: Thu May 17th, 2007 19:36 |
|
John,
Another question that you asked is whether any other Th1 diseases have a gender bias. Actually this has been discussed elsewhere Why do women have more autoimmune disease? and the main bias is toward the female gender in that a number of autoimmune diseases are much more prevalent in females. Also, CFS and FM are more commonly diagnosed in females.
So, the autism sticks out as one of the few that are in the other direction. Ankylosing spondylitis is also more common in males, but I don't recall any other Th1 diseases that are more common in males, unless you count something like heart disease.
Joyce Waterhouse
____________________
20 yrs with CFS/FM/Lyme/IBS, food sensitivities; 1,25D/25D 8/04:64/11 1/05:22/6 9/05:1,25D=12 10/06:22/8, 4/07:25/<4 chewed Ben. 40mg q8h; Mod. P2: 2/23/05, P2: 4/06; P3: 1/1/07
|
tibet
Member
| Joined: | Wed Mar 14th, 2007 |
| Location: | United Kingdom |
| Posts: | 71 |
| Status: |
Offline
|
|
Posted: Fri May 18th, 2007 11:54 |
|
Hi
Dyslexia and Dyspraxia have a gender preference. I have 2 sons.Both have dyslexia and the yponger of the two has Dyspraxia as well.
Stella
____________________
20 years CFS/ME Looking for helpful doctor in UK Have 3 grownup children with disease.
|
Claudia
Member in Phase 3
|
Posted: Sat May 19th, 2007 10:31 |
|
I think we need to remember that CWD infection/Th1 is the Disease - the root cause of variously described "diseases" and syndromes or assortments of symptoms... Whatever you are prone to have go wrong with your body, it will be made worse by Th1 disease. It is not impossible for females to be autistic, but males are more prone to it. If anything, that proves that it is not genetic/sex-linked (or ONLY boys would get it). As I understand it, the XY chromosome thing is a pretty definite thing. You're either a boy or a girl. But having whatever quantities of "male" and "female" hormones is something that is both varied in quantity and proportions in both sexes, hence you get statistical predictability but not set rules. For instance, men can get breast cancer.
So, if you are going to accept that CWD bacteria/Th1 disease is at the root of autism, then that is the disease you are treating, and autism is a symptom (more common in boys.) Maybe mercury makes it worse in brain tissue. Sunshine makes Th1 disease worse for some people. Excess iron made me worse. Stress makes everything worse for most of us! We all experience the disease in a unique way because no two people are exactly alike, not even identical twins.
John, I'd like to know how you are getting along with Jason now. Have you been able to restrict his vitamin D intake? Did you get him NoIR glasses and has he taken to wearing them? Is his physician interested in doing a therapeutic probe?
____________________
MP Phase1 23Mar_06; Phase2 July 10_06; Phase3 Nov 4_06. Dx Thyroiditis (Thyroxine); arthritis; glaucoma; CFS (1988-92);Kidney & bladder probs. Feb06 1,25D=43.3; Aug07 1,25D=27.5; Feb06 25D=44; Aug07 25D=28; Nov07 25D=36; Mar08 25D=16.4
|
Meg Mangin R.N.
Research Team
|
Posted: Sat May 19th, 2007 11:06 |
|
| You can read John's reports of Jason's progress in this thread.
|
Margo
Member Advocate
|
Posted: Sun May 20th, 2007 17:30 |
|
Many Th1 diseases are more often diagnosed in one sex. Ankylosing spondilitis affects 5 times more men than women. Rheumatoid arthritis is more often diagnosed in women than men.
The following link has a chart showing that auto-immune disease (Th1 disease) is much more often diagnosed in women than in men:
http://www.aarda.org/women.html
If autism is found to be a Th1 disease, the numbers of males diagnosed with Th1 diseases will increase.
Sarcoidosis is often discussed in the US as a disease more often found in blacks - but the rates in Scandinavia and in Japan are also relatively high. Since it is a hard-to-diagnose disease, the rates of the disease might reflect the fact that it is looked for more often in certain populations than others.
Many Th1 diseases are hard to diagnose, and many patients receive multiple diagnoses over time. They start out with one diagnosis (ie, psoriasis), then as more symptoms develop, are told that they have an additional diagnosis, such as multiple sclerosis. I would be suspicious of the statistics on the frequency of the diseases because of overlapping symptoms, the absence of a definitive test for many of the diseases, and the frequency of multiple diagnoses. The possibility of physician bias or confusion in the diagnosis should also be considered. Even though a disease might be rare in certain populations doesn't mean it is never found in that population.
Margo
(I mis-typed something, and just corrected it. There are more women with rheumatoid arthritis than men.)
Last edited on Tue May 22nd, 2007 17:22 by Margo
____________________
Parent of teen-aged sarcoidosis/uveitis patient on the MP
|
healingjason
Member in Phase 3
|
Posted: Wed May 30th, 2007 03:21 |
|
I was struck by the following commentary from Meg in another forum:
Inflammation from intracellular bacteria can affect many areas of the brain. Dr. Brian Fallon's borreliosis study (yet to be published) found from SPECT data that the main metabolic changes in the brain were in the region of the parahippocampal gyrus. This area is responsible for receiving sensory input from the outside world, integrating it, and projecting it onto the hippocampus (memory) and Amygdala (fear, aggression, mood).
In the above extract Meg comments on that area of the brain that is '...responsible for receiving sensory input from the outside world, integrating it,.."
This may be just coincidence in the choice of Meg's words or it may be something more but a hallmark of autism (and other childhood developmental disorders) is what is termed 'sensory integration dysfunction' (SID). This is where children are undersensitive or oversensitive to tastes, sounds, textures, smells, indeed anything that involves 'sensing' the world. There is a belief that the brain is malfunctioning in this particular way, as distinct from malfunctioning in acquiring language and understanding social norms and mores, which are the better known features of autism. Indeed, language and social 'deficits' lead to the conventional diagnosis of autism but not SID.
Our Jason is very undersensitive (hyposenstivie) and is very 'hungry' for sensory stimulation - he puts objects in his mouth, loves spicy, hot foods and is very tactile etc. He is given certain physical and other activities to do (jumping, swinging etc) to 'feed' his senses so as to arouse and satisfy him so he can settle and learn and function at school.
As I have noted in my first post, I think the idea of an inflamed autistic brain rather than the existence of structural defects in the brain is fairly well established (Martha Herbert et al). This may also be characterised as underconnectivity between brain regions ands swollen gray matter but I am really guessing when i say this as I have not read this stuff for awhile.
The point of my post is to air a speculation that inflammation from intracellular bacteria could explain a child (or an adult?) with a lot of SID.
John
____________________
Father of Jason, 11 year old autistic boy. Chronic infection evident from urinary amino acids (see bioscreenmedical.com). Benicar 40 mg QW8H from 7 Apr 07. Phase 3 from 28 June 08. Covered up, sunscreen.
|
Current time is 06:54 | Page:   1 2 3 4 5 6 7 8 ...   |
|
|
|
|
