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Grace
Advocate
| Joined: | Tue Sep 14th, 2004 |
| Location: | Australia |
| Posts: | 394 |
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Posted: Thu Mar 6th, 2008 01:28 |
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I'm hardly qualified to post here, but I want to share what I have observe recently and it is of any interests to the MP Researches.
During the last few months with the strong IPR surges I have been having {see my post in PH3 }, I have notice the very first red flag of the impending surges is that I will get a warm, bit of an ache,sensation over my thymus gland. The stronger and longer this lasts, the stronger the IPR will be .This sensation will disappear once the IP is hitting. The sensation only returns just before the next surge.
This has got me thinking about the Thymus gland.Given my limited understanding I've read that the thymus is large when one is born, and continues to shrivel to almost nothing as one ages. Most research is over my head, and is way off about immune and self, pushing a produce, or stem cell reseach. Although one article, that I now can't find which was pushing a product, states that research has shown that those who die from disease, their Thymus is smaller that those who have die suddently from an accident.
Q . For TH1 and MP research purposes, can this change / improvement of the thymus gland be measurable in a blood tests / scan.
Direct analysis of thymic function in children with Down's syndrome..
Background
Down's syndrome (DS) is characterized by several immunological defects, especially regarding T cell compartment. DS is considered the best example of accelerated ageing in humans. Direct observations of the thymus have shown that in DS this organ undergoes severe histological and morphological changes. However, no data on its capacity to generate T cells are present in the literature............
http://www.immunityageing.com/content/2/1/4
Importance of thymus derived rosette forming ce;lls in pulmonary tuberculosis
[url=http://lrsitbrd.nic.in/IJTB/Year 1993/July 1993/jul1993 G.pdf]http://lrsitbrd.nic.in/IJTB/Year%201993/July%201993/jul1993%20G.pdf[/url]
Last edited on Thu Mar 6th, 2008 07:39 by Grace
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CFS, oct04 Ds=26/48, 4/07=7/24 MP PHI 2/05, PhII 6/05, PhIII 6/06, beni Q6H, Noirs rarely inside no nature light low watt, outside 40%10%, Hat, all covered, no gloves, paracetamol
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Joyful
Research Team
| Joined: | Sat Jun 9th, 2007 |
| Location: | West Coast, USA |
| Posts: | 531 |
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Posted: Thu Mar 6th, 2008 07:13 |
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hello grace,
I'm as in over my head discussing this as I can be... but here is an observation of my own to share:
I have no awareness of tightness in my chest during my usual daytime activities. However, when I take a shower and the warm water hits my chest about where the thymus area should be, I seem to experience this deep sigh and start breathing more freely in my upper chest. It feels like something good is happening, so I let usually just stand there and take some deep breaths.
I have no clue why this is, but just thought I'd add it to your ponderings.
Last edited on Thu Mar 6th, 2008 07:19 by Joyful
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Lyme Babs Bart - 125D50 Ph1Jul07 ModPh2Sep07 Ph2Feb08 Ph3Aug08 - cal/mag lysine hydroxyzine valium - rarely leave house NoIRs cover up low lux home - 25D17 Jul08
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Goliad
Health Professional
| Joined: | Mon Aug 6th, 2007 |
| Location: | Temple, Texas USA |
| Posts: | 5 |
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Posted: Tue Mar 11th, 2008 18:10 |
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Isn't the thymus gland where the all important T3 lymphocytes that initiate the defective signals to B-cell lymphocytes are developed and distribute throughout the body? ( or something to that effect?). I have always thought the T-cell and B-Cell lymphocytes are the greatest culprits where the invading L-forms, or mycoplama wreak their damage by altering the perception of T-cell,and B-cell lymphocytes to develop abnormal antibody responses that are the hallmark of serious autoimmune development?
Overall, isn't the bottom line this deviation from normal lymphocyte antibody formations againest "self" perceived abnormally as antigens? This was the early basis for an early pioneer to initiate thymus drainage of newly formed lymphocytes to lessen abnormal antibody reactions. ( Or so I understood the Road Back folks.) The whole problem with the L-form alteration of DNA or other celllular functions of the lymphocytes seems to be the terrible destruction that is influenced to occur with such infectious invasions of these blood cells.
Last edited on Tue Mar 11th, 2008 18:16 by Goliad
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Ignorance is preferred over knowledge pursuits in the interests of money. (The USA Doctor's creed)
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Dr Trevor Marshall
Research Team
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Posted: Tue Mar 11th, 2008 18:42 |
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The lymphocytes have little impact on chronic disease, except to the extent that they themselves are parasitized by the bacteria. This is the reason that Medicine has failed to understand these idiopathic diseases. They have focused on Lymphocytes and Natural Killer cells, whose behavior is secondary to the primary infectious process.
If you look at a Sarcoid granuloma under a microscope, it is 90% monocytes and immature macrophages. The lymphocytes are expelled to the periphery. I would have thought that somebody, somewhere, would have understood the importance of this, long before I started to figure out the disease process. But no...
As for the location of idiopathic pains, etc, lymph nodes are located all over the body:
and these should be considered the prime suspects to swell and produce pain from immunopathology in Th1 disease.
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Dr Trevor Marshall
Research Team
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Posted: Tue Mar 11th, 2008 18:47 |
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Grace,
One of the genes known to be transcribed by the VDR is located in the region known to be responsible for Down Syndrome. The function of this gene is currently unknown, but it certainly seems as though Th1 pathogens might well be involved in this most classic of "birth defects."
ps: interesting that the incidence of DS increases with the age of the mother. Another pointer to Th1 involvement.
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