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Posted: Sun Oct 31st, 2004 02:03 |
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Why doesn't the MP use some of the other antibiotics?
Many commonly used antibiotics are beta-lactams. Beta-lactam antibiotics include penicillins, cephalosporins and related compounds. These antibiotics work by inhibiting the cell wall of bacteria and are not effective against the Cell Wall Deficient (CWD) bacteria that cause Th1 inflammation.
The stealth bacteria, which we are fighting, are "cell wall-less" variant forms of the original parent bacteria. Cell Wall Inhibiting (CWI) antibiotics don't get rid of them. In fact, CWI antibiotics just help to create more of them.
Beta-lactams have been shown to actually cause Cell Wall Deficient (intracellular bacteria) to form from blood-borne bacteria.
Please see:
http://tinyurl.com/572yt
and
http://tinyurl.com/6b3lv
Here are some of the Dienes' papers (cited by Klienberger-Nobel) which represent the earliest warning that Beta-lactam antibiotics, penicillins and cephalosporins (such as Rocephin) actually promote the formation of the persistent L-forms when they kill the more easily visible walled-bacteria
The Transformation of Typhoid Bacilli into L Forms under Various Conditions
http://www.pubmedcentral.gov/picrender.fcgi?artid=385825&blobtype=pdf
The Isolation of L Type Cultures from Bacteroides with the Aid of Penicillin and Their Reversion into the Usual Bacilli:
http://www.pubmedcentral.gov/picrender.fcgi?artid=518606&blobtype=pdf
Transformation of bacteria into L forms by amino acids:
http://www.pubmedcentral.gov/picrender.fcgi?artid=169421&blobtype=pdf
The MP goal is to kill these CWD (divergent) bacteria, even if it means experiencing the temporary discomfort of immune system "die-off" reactions. For this reason, different antibiotics known as "protein synthesis inhibitors" (PSI) are selected for the MP to assist the immune system in eliminating these sneaky, intracellular pathogens once and for all.
Safety
"We only use abx in the MP which are known to be safe, and have a long track record (mino is nearly 40 years) of safety. They also have very limited ability to encourage resistant bacterial strains. There is no point in looking at any other antibiotics, as, even if the other abx work, the proven ones which we already have available will provide a faster rate of bacterial killing than most people can tolerate."
..Trevor..
IV antibiotics
If you were administered IV antibiotics while the benicar had activated your innate immunity, so many intra-cellular bacteria would be killed that you would probably die from the immunopathology. That is not a desirable outcome.
The reason we use low dose, oral, antibiotics is to control the rate of killing of the bacteria to a level that the body can handle.
I did speak to an anesthesiologist that used IV clindamycin as an antibiotic in a hospital environment, and he had already noticed that folk who were using ARBs were particularly sensitive, and had adverse reactions. They were using less-effective ARBs than Benicar.
The MP is a total entity. Every bit of the science has been optimized. Any changes introduce risk. I would be happy to send Doc a copy of the "Science" DVDs if it would help him/her understand that.
..Trevor..
Palliation
Some folks report that the use of CWI antibiotics makes them feel better. But for folks with Th1 inflammation, this improvement is temporary. There is a big difference between feeling better and actually getting better.
The increase in CWD bacteria makes it counterproductive to take CWI antibiotics just because it makes you feel better temporarily.
It's to your advantage to bite the bullet and choose to endure the immunopathology now, in order to feel better later, than it is to deceive yourself into believing that there is a short-cut to healing and ultimate recovery, by expecting to take an antibiotic just because it gives you immediate, but only temporary, relief very quickly.
Adverse reactions to beta-lactam antibiotics
The beta-lactam antibiotics protect CWD bacteria from the immune system. But they also kill any walled bacterial organisms that may be present. If you have not been taking regular antibiotics you may be carrying both active bacteria (those with cell walls) and CWD (stealth bacteria) in your bloodstream and other tissues.
Patients who experience side effects, 'allergies' or adverse reactions while taking these CWI antibiotics are probably experiencing immunopathology resulting from the beta-lactam antibiotics killing active bacteria, not the CWD forms.
This does not mean that a person with inflammatory symptoms does not have CWD bacteria since the CWD can revert back to walled bacteria from time to time. Although unlikely, an immune system reaction to a CWI (beta-lactam) antibiotic is always possible.Last edited on Tue Aug 28th, 2007 09:08 by
____________________
*We can help you understand chronic disease, but only your physician is licensed to give you medical care *
Always consult your physician before commencing or changing any treatment he/she has prescribed for you
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Dr Trevor Marshall
Research Team
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Posted: Sun Oct 31st, 2004 09:44 |
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Rocephin
Unfortunately, IV Rocephin often results in relapse when the treatment is discontinued. Rocephin is a beta-lactam antibiotic (see post above) and it has a purely palliative action, and actually protect the Th1 pathogens.
The 5-yr study at Columbia under Brian Fallon found that chronic Lyme patients' symptoms improved during IV Rocephin, but relapsed upon withdrawal, and all improvement except some muscle tone had disappeared within 3 months of cessation of the beta-lactam.
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Posted: Sat Jul 30th, 2005 22:19 |
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link
FDA action will encourage increase in L-form bacteria
FDA bans Bayer antibiotic for poultry use
"I agree with the FDA's decision, except that I know that it opens up a can of worms that the FDA doesn't even begin to understand - the epidemic spread of zoonotic bacteria (zonnosis = animal to man).
The L-forms can survive pasteurization and some cooking. It will probably take a number of years before the effects of this FDA decision start to be seen as INCREASED human disease, rather than decreased, IMO. But it is a necessary step in the process of scientific learning 
The MP uses bacteriostatic antibiotics, which generally do not create as much problem with resistance as the bactericidal antibiotics in common use in medicine, such as the flouroquinolone banned by the FDA in poultry.
...the use of Beta-lactam antibiotics (Peniciillin, Ampicillin, Cephalosporin) are one of the driving forces behind the current epidemic of Th1 diseases. Even Rifampin (used in TB and Leprosy) encourages the formation of L-forms when it kills the blood-borne bacteria.
The ID specialty has to learn how antibiotics really work, and how to formulate combinations which kill both the blood-borne and L-forms of the infection. I fear this is going to take another decade or two...
..Trevor..
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