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Every week brings more bad news for Vitamin D
 Moderated by: Prof Trevor Marshall Page:  First Page Previous Page  ...  7  8  9  10  11  12  13  14  15  16  17  ...  Next Page Last Page  
 

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be-well
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 Posted: Wed Dec 23rd, 2015 23:13

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although the GP insisted on giving my elderly 90 yr old mother  vitamin d3 with calcium in a liquid drink first thing every morning after a wrist fracture, for 10 years, her vitamin d levels measured 25d-5, 1,25d, 70.  She had dementia and alzhiemers, her gait was slightly uneven, and the GP simply blew me off with a comment about the elderly 'not getting sufficient sunlight, because they live in a nursing home".

I actually felt powerless to defend my lovely mum and I wish this awful issue would get sorted.




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mcs,chronic fatigue,hiatus hernia,grains intolerance,tinnitus,photosensitivity.
25-D-3.3,-1,25D-70,Dec 2014.25-D-18, 1,25D,45.Sept 2019.
HeatherZ
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 Posted: Sat Dec 26th, 2015 21:28

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It is very sad be-well :(



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MP start Aug'11 (no breaks) | Dx - none | Trichotillomania, GI problems, urinary dysfunction, sleep disturbance, anxiety| 25D= 6ng/ml May'12, 10ng/ml Jan'13, 8ng/ml Aug 13, 8ng/ml Feb'14, 5ng/ml July'15, 7.2ng/ml Jan'19 (Break commencing Aug 2017 to have
Sallie Q
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 Posted: Sat Dec 26th, 2015 22:32

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i today heard a recorded talk by another (now deceased) Oz scientist who mid last century demonstrated that
treatment for rickets - vit. D supplement for that (extremely rare in modern society) condition
 and
treatment for osteopenia - calcium (when deficient in that element)
 must be completely different.

His research was unpublished for quite some time because it did not fit the paradigm, nevertheless, the information has been around since Prof. Marshall was in junior school

Modern doctors are over-worked or plain lazy and/or badly educated :(

Last edited on Sat Dec 26th, 2015 22:50 by Sallie Q



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VideoMP'08-'11(now@3x20mgOLM)Dx Depressn'70,node&brCancer'90,BCC'05,Sjgren08|SxCFS,RA,stroke,ASD,reflx 25D=9ng/ml_Nov'17Info
Bane
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 Posted: Mon Jan 4th, 2016 23:08

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Taking vitamin D may benefit people with multiple sclerosis

http://www.sciencedaily.com/releases/2016/01/160104080559.htm
http://www.neurology.org/content/early/2015/12/30/WNL.0000000000002316

"The people taking the high dose had a reduction in the percentage of inflammatory T cells related to MS severity, specifically IL-17+CD4+ and CD161+CD4+ cells. When the increase in vitamin D levels in the blood over base line levels was greater than 18 ng/ml, every additional 5 ng/ml increase in vitamin D led to a 1 percent decrease in the percentage of IL-17+CD4+ T cells in the blood"

"This study provides Class I evidence that cholecalciferol supplementation with 10,400 IU daily is safe and well-tolerated in patients with MS and exhibits in vivo pleiotropic immunomodulatory effects"

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 Posted: Mon Jan 4th, 2016 23:16

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Higher monthly doses of vitamin D associated with increased risk of falls

http://www.sciencedaily.com/releases/2016/01/160104125326.htm

"Doses of 60,000 IU and 24,000 IU plus calcifediol were more likely to result in 25-hydroxyvitamin D levels of at least 30 ng/mL but they were associated with no benefit on lower extremity function"

"Compared with a monthly standard-of-care dose of 24,000 IU of vitamin D3, two monthly higher doses of vitamin D (60,000 IU and 24,000 IU plus calcifediol) conferred no benefit on the prevention of functional decline and increased falls in seniors 70 years and older with a prior fall event"

Nyima
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 Posted: Thu Jan 7th, 2016 12:26

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http://www.independent.co.uk/life-style/health-and-families/features/the-sun-goes-down-on-vitamin-d-why-i-changed-my-mind-about-this-celebrated-supplement-a6800191.html



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MP start Dec 10 (2 mo. break Jan-Mar '11) | Multinodular thyroid | frequent illness| fatigue | brain-fog | gluten intolerance (poss coeliac), eczema with dairy products | 25D (ng/ml): 4(14 Apr 11), 5.3(26 Apr 13), 4(Jan 14), 5.5(July 14)
scooker48
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 Posted: Thu Jan 7th, 2016 17:00

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Good find, Nyima.:)



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D25, Total: 12 measured 11/3/15 Started MP=01/04/05 Diagnosis: Sarc 12/04; "cat scratch disease" or necrotizing graunulomas 10/88; Raynaud's (diagnosed 1980?)
Trudy.Heil_NP
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 Posted: Fri Jan 29th, 2016 18:50

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No Effect of High-Dose Vitamin D Treatment on β-Cell Function, Insulin Sensitivity, or Glucose Homeostasis in Subjects With Abnormal Glucose Tolerance: A Randomized Clinical Trial

http://care.diabetesjournals.org/content/early/2016/01/07/dc15-1057.abstract

"CONCLUSIONS This study gives no support for any substantial effect of high-dose vitamin D treatment for 8 weeks in prediabetes or diet-treated type 2 diabetes on β-cell function, insulin sensitivity, or glycemic control." :D

Onward & Upward,
Trudy :cool:



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MP June08(NoBreaks)Ph1June28-08 ModPh2Aug11-08 Ph2Sept25-08 Ph3Oct4-10 Benicar Only 2012|VitD1,25 6-08:72; VitD25:12-15 12.0 |FMS40+yrs CystAcne65 EBV66 ClsdHdInjry66 PCOS71 Hashmtos81 HSV2&6 81 TotlHyst86 Endmtrs PsorFeet95 Strep&ClamydPneumx3
Joyful
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 Posted: Sat Jan 30th, 2016 05:50

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Exposure to excessive oral vitamin D in youth: a risk factor for celiac disease in later life?

Abstract

Celiac disease is an autoimmune disease initiated by an allergic reaction to gliadin a component of the protein gluten that is found in wheat and other grains.

Vitamin D is a prohormone with a number of biochemical functions including immunomodulatory functions in its active form.

Supplementation with large doses of vitamin D induces symptoms that are similar to celiac disease in some of the population.


Some researchers have noted that consumption of large doses of oral vitamin D early in life is associated with increased incidence of allergic diseases later in life.

An examination of celiac disease suggests that an allergy is fundamental to its development and it features Th2 cytokine elevations which typically characterize allergic diseases.

In addition it has comorbidity with allergic diseases where exposure to large doses of vitamin D early in life is a risk factor. This raises the possibility that large doses of vitamin D early in life could be a risk factor for inducing celiac disease later in life as well.

The comorbidity between Williams syndrome, which is characterized by spikes in blood levels of vitamin D, and celiac disease gives additional credence to this hypothesis. Epidemiological evidence supports this hypothesis as well. Vitamin D consumption among the young and celiac disease prevalence are high in Sweden, Finland, and the United States. Vitamin D consumption among the young and celiac disease prevalence are low in Russia and Germany.

The timing of the increases in prevalence in the United States and Sweden also coincides with increasing vitamin D consumption. This is most striking in Sweden where a law required significant vitamin D fortification of food starting in 1983 and the start of the celiac epidemic in Sweden has been independently dated from 1984.

This hypothesis also provides a potential explanation for some seemingly contradictory results in the literature regarding breastfeeding and risk of celiac disease.





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Lee
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 Posted: Wed Feb 3rd, 2016 20:06

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Did anyone else see the program by 20/20 News on PBS, that was on the hidden dangers of supplements?  It just scratched the tip of the iceberg but it did mention "D" and other vitamins and supplements that are clearly dangerous!  Lee



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MP-Feb05 ABX Breaks 7mth,18mth,3yr,5yr 2003 Sarc, Hyper-flex FM, Hashimotos, COPD, skin, joint pain and Glaucoma. 2014 blood work normal D-levels 4-5.
Lee
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 Posted: Thu Feb 4th, 2016 20:56

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Here is more info on the program I mentioned on PBS about the dangers of supplements.  Lee
https://www.youtube.com/watch?v=dCb5R4YVf2c



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MP-Feb05 ABX Breaks 7mth,18mth,3yr,5yr 2003 Sarc, Hyper-flex FM, Hashimotos, COPD, skin, joint pain and Glaucoma. 2014 blood work normal D-levels 4-5.
scooker48
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 Posted: Tue Apr 26th, 2016 20:24

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This one looks interesting; at least it refutes the low D25 myth.

http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0064348

Serum 1,25-Dihydroxyvitamin D: An Outcome Prognosticator in Human Sepsis
AbstractIn sepsis, the vitamin D active metabolite 1,25-dihydroxyvitamin D (1,25(OH)2D) may play a crucial role by its action to produce cathelicidin and improve endothelial barrier function, such that a deficiency in 1,25(OH)2D is associated with poor outcome. To test our hypothesis, we performed analysis of stored plasma samples from a prospective observational study in 91 patients with sepsis, age of 59.1+/−2.0 years, 52.7% females, and 11.0% deaths at 30 days. Vitamin D status, including 25-hydroxyvitamin D (25(OH)D), 1,25(OH)2D, 24,25-dihydroxyvitamin D (24,25(OH)2D), and parathyroid hormone (PTH), were measured daily over 3 days after hospital admission. At baseline, 1,25(OH)2D was significantly different between survivors vs. non-survivors. But there was no significant difference in 25(OH)D, 24,25(OH)2D, and PTH. In a multivariable binomial logistic regression model, age, total calcium and 1,25(OH)2D were significant predictors of 30-day mortality. Kaplan Meier analysis showed that patients with mean 1,25(OH)2D measured over 3 days of < = 13.6 pg/mL had 57.1% 30-day survival compared to 91.7% in patients with 1,25 (OH)2D level >13.6 pg/mL (p<0.01). From repeated measures regression analysis, there was significant increase in 1,25(OH)2D for increases in 25(OH)D in both survivors and non-survivors. However, compared to survivors, the low 25(OH)D in non-survivors was insufficient to account for the larger decrease in 1,25(OH)2D, indicating a dysfunctional 1α-hydroxylase. Additionally, there was a significant negative correlation between PTH and 1,25(OH)2D in both survivors and non-survivors, suggesting a severe impairment in the effect of PTH to increase renal 1α-hydroxylase activity. In conclusion, low 1,25(OH)2D levels are associated with increased 30-day mortality in sepsis patients, likely due to impaired 25(OH)D hydroxylation and PTH insensitivity. Our data also suggest that the active metabolite 1,25(OH)2D may be an important therapeutic target in the design of sepsis



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D25, Total: 12 measured 11/3/15 Started MP=01/04/05 Diagnosis: Sarc 12/04; "cat scratch disease" or necrotizing graunulomas 10/88; Raynaud's (diagnosed 1980?)
bookdad
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 Posted: Thu Apr 28th, 2016 18:50

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I just contacted Imagine Foods on their web site and thanked them for continuing to make Rice Dream Classic.

Last edited on Thu Apr 28th, 2016 18:59 by bookdad



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bookdad
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 Posted: Thu Apr 28th, 2016 18:50

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I have been using Rice Dream for some time now. About a year and a half ago they came out with an "Enriched" version. it seems that the health food stores, Walmart, and suppliers are pushing to replace the "classic" version (no vitamin D) with the Enriched. Consequently, the shoppers are raiding the shelves as soon as the classic is put out. The enriched is just sitting there. When will they get a clue? Not many of us want all that added crap. Walmart has since discontinued offering the classic version. When I asked about it they said they cant get it anymore. I promptly went to the health food store and ordered 2 cases (very long shelf life) and received them in a week. somebody isn't telling the truth. Also of interest is the ubiquitous soy. its in everything. this is a really bad additive for men (mimics estrogen) and for women if you have too much estrogen you grow extra yeast. so, if we are going to write the food producers, lets include a laundry list, or better, just ask for pure food.



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Sarcoidosis/lungs RA Ph1Nov05 Ph2Mar06 Ph3Aug06 lite exp r/t to work cover up NoIRs Ph5 Aug 09
Cairo123
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 Posted: Thu Apr 28th, 2016 22:05

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This is a classic result of eating processed foods. When one consumes processed foods versus real food (whole unprocessed food) one is at the mercy of whatever the manufacturer decides to put in it. If it has a list of ingredients it is probably processed.

http://mpkb.org/home/food/additives

http://mpkb.org/home/food



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Sarcoidosis, Arthritis, Fallen Arch, Vascular Insufficiency, fatty liver,diverticulosis, 1,25D 16 (Oct08) | Ph1 Nov07, Ph2 Jan08, Ph3 May 09 Ben Only: Jun 10- Sep 10|Break Sep 10-Dec 10|Resume MP Dec 10|
bookdad
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 Posted: Tue May 10th, 2016 15:53

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thank you those were very helpful links. I've sent them to my friend Brad that is just starting. I read all the ingredients on every thing and my sweetheart is even doing that too to make sure we don't get something with hidden sources of antagonists. I was so surprised at how many things have added Soy. Even "natural" cheese!!!



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Sarcoidosis/lungs RA Ph1Nov05 Ph2Mar06 Ph3Aug06 lite exp r/t to work cover up NoIRs Ph5 Aug 09
bookdad
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 Posted: Tue May 10th, 2016 15:53

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read the ingredients!

Last edited on Tue May 10th, 2016 15:55 by bookdad



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 Posted: Tue May 10th, 2016 20:57

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Just reading the ingredients is not enough if the product has any dairy, processed or otherwise, in it, as manufacturers that use fortified products as ingredients do not generally mention the fact that any of their ingredients are fortified, only if they add the D themselves.

Cynthia



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MP start 10/08,break 1/16 - 9/16, Spondylitis'97,early Diverticulosis'98,early AMD'08,Calcium anomaly'95,TypeII Diabetes(?)'02,Degenerative hip disease'12, 25D=10.8 May'18 (preMP 125D/25D=47/43) https://marshallprotocol.com/forum30/13911-2.html
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 Posted: Wed May 11th, 2016 06:14

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This gets back to the very problem. If it has ingredients it is probably processed. It is best that one eat as much whole unprocessed food as possible that one prepares from scratch. That way you know and can control what goes into it.



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Sarcoidosis, Arthritis, Fallen Arch, Vascular Insufficiency, fatty liver,diverticulosis, 1,25D 16 (Oct08) | Ph1 Nov07, Ph2 Jan08, Ph3 May 09 Ben Only: Jun 10- Sep 10|Break Sep 10-Dec 10|Resume MP Dec 10|
Claudia
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 Posted: Mon May 16th, 2016 16:09

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Indeed. Jamie Oliver said it best.
"Real food doesn't have ingredients, real food is ingredients."



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MP Phase1 23Mar_06; Phase2 July 10_06; Phase3 Nov 4_06. Dx Thyroiditis (Thyroxine); arthritis; glaucoma; CFS (1988-92);Kidney & bladder probs., Osteoporosis Dx 2015, Burning Mouth Syndrome! 2016. Feb06 1,25D=43.3; Aug07 1,25D=27.5; Feb06 25D=44; Mar08

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