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What I learnt from talking with Rolf Zinkernagel
 Moderated by: Prof Trevor Marshall Page:  First Page Previous Page  1  2  3  4  Next Page Last Page  
 

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Prof Trevor Marshall
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 Posted: Sun Jul 30th, 2006 13:08

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Sherry,
Viruses typically have small genomes, say 70,000 base-pairs, while bacterial genomes typically span several million base-pairs.

There are many other differences too:):)

Steve
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 Posted: Mon Jul 31st, 2006 17:35

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     On twenty-first century Earth, what was typical can no longer be taken for granted. Myriad novel life forms are possible if able to combine parts from viruses and bacteria......and who-knows-what else (fungi, nematodes, arthropods?).....whether accidental or engineered.:shock: 

     To go back to the beginning again, from the information presented; it appears the mice brains present a different pathogenesis model than the mice bodies when infected with the same virus. This makes sense. "Life" is as diverse as it is tenacious. Using Darwin's platform, the lifeform which benignly parasitizes and lives symbiotically with a host has a greater advantage for survival and proliferation than a more destructive invader. This virus has hit on a winning set of mutations that guarantee the survival and proliferation of itself and it's host. For the most part, these lifeforms (or parts thereof) are smaller than most have bothered to look. We should expect there to be a thriving, diverse, and and storied history of infectious agents of this type. Especially in the reproductive and nervous systems. Call them L-forms, CWD or pleomorphic bacteria, "self-determining" DNA/RNA pieces, viruses................this world is truly their oyster! :dude:

     Drs. Blaney and Marshall: Whether part of the innate or adaptive immunity systems, how does the body get rid of infectious agents which are sequestered inside living host cells on the MP? Do these pathogens get induced to leave the cell, leaving the host cell alive? Or are the host cells killed along with the pathogens? What are the specific sequence of events which take place, on the cellular level?



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 Posted: Mon Jul 31st, 2006 17:42

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Steve
You may be interested in this Information:
CELL WALL DEFICIENT BACTERIA AND THE MARSHALL PROTOCOL
Thanks, Barb ...



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Rosie
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 Posted: Mon Jul 31st, 2006 20:18

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Infants vaccines make me nervous.  Which vaccines would you suggest for babies and in what order? My first grandchild is coming soon and if some of what my family members have gone through could be prevented I would be grateful.

Thanks, Rosie



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 Posted: Mon Jul 31st, 2006 23:18

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Rosie,

The question of whether or not to vaccinate is fraught with controversy. We cannot advise on particular vaccines. Parents should study the issues and decide for themselves.

Vaccines are given in a certain pattern to promote their effectiveness. The baby's pediatrician or the public health office are two resources to ask how long immunizations can be delayed and how far apart they can be spaced.

Best,

Meg

Frans
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 Posted: Sat Aug 12th, 2006 04:16

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Trevor, another question I have that relates loosely to this thread:

I understand from another thread that you have been busy with molecularly 'shooting' Beni's molecule at several bacterial genomes with success.

Would it be perhaps interesting to do the same with viral genomes? Looks to me that might be an interesting exercise.

Sincerely, Frans



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Fara
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 Posted: Mon Aug 14th, 2006 21:39

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This thread is very interesting to me.  I don't ever remember a tickbite but I did get sick right after my Rabies Vaccine (required in veterinary school).  My mother also had Bell's Palsy when she was pregnant with me, but my identical twin sister is fine.  She doesn't get vaccinated for anything...



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MPolson
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 Posted: Sat Aug 19th, 2006 12:42

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This thread has got me thinking (I haven't been doing alot of that lately).

My first thought is that I am considered a hepatitis B carrier. This was discovered after I got the first two out of three hep B vaccinations. Could I have actually developed the disease from the vaccination if my immune system didn't work properly? I say that I am considered a Hepatits B carrier because I test positive for the antigen but I test negative for the hepatitis B DNA.

Secondly, since I am considered a Hep B carrier, both of my children were vaccinated the day they were born. Could this have been a big mistake? But if they now test positive for Hepatitis B antigen, would that indicate that thier immune systems were able the convert it? Maybe because I breast fed them, the innate immunity was given to them throught the breast milk.

Thirdly, you had discussed earlier about the transmission of bacteria and viruses through the placenta. I believe that this has been speculated in mothers that are HIV positive. This is why it is so highly recommended that these mothers take certain antiviral medications during the pregnancy. If the threat was only due to transmission at birth, they would just have to give them the treatment starting at the time of the exposure(during delivery) just as they do anyone who has an HIV exposure. I don't know if placental transfer has been proven in HIV pregnancies, but they do treat it as if it were.

If HIV can transfer through the placenta, isn't it pretty likely that other bacteria do as well?

MPolson



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tickbite
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 Posted: Sat Aug 26th, 2006 16:56

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Since this thread talks a lot about vaccines I thought I would post here. Feel free to move it somewhere more relative.

Scientists Identify Immune System Trigger For Fighting Lyme Disease

News article dated August 23, 2006

Anyway, so what? They've found a glycolipid which triggers an immune response from the body's natural killer (NK) T cells. Surely this isn't the only receptor that borrelia communicate with. How on earth would this help?:? I guess these guys are just behind the times still. They've figured out a wierd way in which a parent form stimulates T cells. Whoopee doo.



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Prof Trevor Marshall
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 Posted: Sat Aug 26th, 2006 17:09

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greg,
Every young biochemist who graduates these days is trying to make a million - by discovering a drug that can be commercially marketed. They are typically given 0.1% (or so) of the profits - and so their life goal becomes to try and fit disease into the constraints of commercial drug discovery.

I am sure these folks are thinking about a vaccine, or something they can market.

Of course, it won't work. That is why we keep getting promises for our research tax dollars, and not results. Meanwhile, we keep giving them money...

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 Posted: Mon Nov 13th, 2006 17:58

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Hi,

I was trying to read a bit of Dr. Z's paper, and I think I asked this before ( not sure), but why does he always refer to autoimmune diseases as being caused by viruses? Do you think there are any viral components to any cases of sarcoidosis or other Th! inflamatory diseases?

Thanks,

Norman

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 Posted: Tue Nov 14th, 2006 10:59

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I was wondering myself. Here is what Dr Marshall wrote in a paper

Antibiotics in Sarcoidosis - Reflections on the First Year (8-3-03)
http://www.joimr.org/phorum/read.php?f=2&i=38&t=38

The SARS Coronavirus is a pathogen with the apparent ability to virulently hyper-activate the immune system in this manner [9,10,11,12]. While the granuloma of sarcoidosis are formed by an accumulation of considerably less virulent pathogens than SARS, the anomalous T-cell Receptor alpha-beta V protein is similarly present [13].

Last edited on Tue Nov 14th, 2006 11:00 by wrotek



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MarkN
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 Posted: Mon Nov 27th, 2006 16:54

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Speaking of vaccines I know two people whose health was never the same after having adult vaccinations.



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 Posted: Tue Nov 28th, 2006 08:52

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HI MARKN

This is Fred in WV.  Mark, please tell us more.

Remember, we are all in this together and I am pulling for us.

Your friend in sarcoidosis

Freddie



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MarkN
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 Posted: Tue Nov 28th, 2006 15:47

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Hi Freddie

One man was very athletic in his 40's and had Hep. booster and some other shots for going overseas. He got a rash and noticed his swimming and biking endurance suddenly went down. He never used to get sick but for the past two winters has had walking pneumonia. Also an older woman, already in poor health but getting worse, and her dementia seems to have doubled overnight after some shots. So no vaccines for me - who knows the risks involved between the viruses, bacteria, mercury etc.



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Prof Trevor Marshall
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 Posted: Tue Nov 28th, 2006 16:35

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Mark,
The mercury is not a factor. Mercury has become associated with these chronic diseases because the body cannot clear heavy metals when the immune system becomes overloaded with Th1 pathogens. But once patients recover on the MP they no longer exhibit elevated mercury or lead levels. So mercury is just another "co-infection," if you like, just like Babesia and the other things that are often observed with Th1 disease, but are not causative, just 'associated.'

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 Posted: Wed Dec 13th, 2006 06:59

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Here's an interesting article about a study in Germany. After thinking about it, it appears that it really, really makes sense if one follows Dr. Marshall's and Rolf Zinkernagel's studies/ideas about how early infection affects one's ability to respond when re-infected later in life.

Younger Siblings May Boost Brain Tumor Risk



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 Posted: Fri Jan 5th, 2007 07:12

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Trevor, something just hit me regarding infected stem cells.

If innate immunity is suppressed from birth on and babies start gathering infections, it would only be logical to assume the stem cells get infected also, since I would think they are also wholly dependant on innate immunity ?

Sincerely, Frans



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Prof Trevor Marshall
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 Posted: Fri Jan 5th, 2007 07:15

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That is correct, Frans.

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 Posted: Fri Jan 5th, 2007 07:28

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Trevor, you once stated here on the forum that the world wasn't ready yet to face the consequences of vitamin D being immunosuppressive.

I understand fully now...

It's mind boggling really, this will shake the world of medicin to its core once the meaning of this sinks in...

wow....



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