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My AAEM 2006 presentation is on YouTube
 Moderated by: Prof Trevor Marshall Page:  First Page Previous Page  1  2   
 

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NorCalJim
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Joined: Thu Aug 24th, 2006
Location: Sacramento, California USA
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 Posted: Fri Nov 30th, 2007 10:55

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The google video doesn't seem to be working for me anymore. Anyone else having trouble with it?

NorCalJim



____________________
Ankylosing spondylitis Ph1Mar07 Ph2Jun07 Ph3Nov07, no other meds or supps 25D18 (May07) 25D16 (Feb08) 25D16 (Jun09) NoIRs in/out cover up out
Prof Trevor Marshall
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Joined: Fri Jul 9th, 2004
Location: Thousand Oaks, California USA
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 Posted: Tue Dec 18th, 2007 11:14

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I have now figured out how to put up longer video segments on YouTube, although the quality is a little lower than the original 8-segment version.

The new video is in two 35-minute segments at URLs:

http://www.youtube.com/watch?v=RpocxjKJxag
http://www.youtube.com/watch?v=ZMn-zUTTHgw

and these  are now the best URLS to use when you are sharing the video with your friends. Please update your bookmarsk.
 

Phillyguy
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 Posted: Sun Aug 22nd, 2010 16:12

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Dr Trevor Marshall wrote: Depends what caused the birth defect.
It seems that Vitamin D metabolism abnormalities (resulting from Th1 disease) are associated with pre-eclampsia. The VDR is also associated with the transcription of many genes, including assocuiated with Down Syndrome, which it is hard to "go in and fix." However, infections passed from mother to child can be treated. There is much still lto learn.However, which condition specifically are you concerned about?
 


An immunological insight into the origins of pre-eclampsia (10/2010)

BACKGROUND: Pre-eclampsia is a syndrome of heterogeneous origin characterized by deficient placentation due to the inability of the cytotrophoblast to acquire an invasive phenotype and to remodel the uterine spiral arteries. One of the main problems observed early in pre-eclampsia is an altered regulation of the immune system, where the shift toward a Th2 cytokine profile observed in normal pregnancies, does not occur. In pre-eclampsia, high interferon (IFN)-gamma concentrations are present, along with transforming growth factor-beta cytokines, which retard migration of cytotrophoblasts.

METHODS: A review of the scientific literature was performed on the immunological factors associated with the origins of pre-eclampsia. The various components of the immune system that may be participating in the aberrant immune activation that pathologically affect early pregnancy events and inhibit cytotrophoblast invasion were identified.

RESULTS AND CONCLUSIONS: Cells and their signaling and regulatory molecules have been implicated in the immunological alterations found in the placental microenvironment of patients who develop pre-eclampsia. One of the main differences found in pre-eclampsia is a shift toward Th1 responses and the production of IFN-gamma. The origin of IFN-gamma is not clearly identified and could be the uterine natural killer cells, the placental dendritic cells modulating Th responses, alterations in synthesis of or response to regulatory molecules, or changes in the function of regulatory T cells in pregnancy. Aberrant immune responses promoting pre-eclampsia may also be due to an altered fetal allorecognition or to inflammatory triggers. Understanding the immunological basis for pre-eclampsia will expand knowledge regarding other adverse pregnancy outcomes.

http://www.ncbi.nlm.nih.gov/pubmed

 


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